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@berserk

17 Hours ago

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@berserk

17 Hours ago

Aenean vulputate eleifend tellus. Aenean leo ligula, porttitor eu, consequat vitae, eleifend ac, enim. Aliquam lorem ante, dapibus in, viverra

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@berserk

17 Hours ago

Aenean vulputate eleifend tellus. Aenean leo ligula, porttitor eu, consequat vitae, eleifend ac, enim. Aliquam lorem ante, dapibus in, viverra

T4

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Immunoenzymatic colorimetric method for quantitative determination of thyroxine (T4) concentration in human serum and plasma. T4 ELISA kit is intended for laboratory use only.

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PAPILLOMAVIRUS TYPE 18 (HeLa cells) DNA Ctrl
PAPILLOMAVIRUS TYPE 18 (HeLa cells) DNA Ctrl
  • Papillomaviruses are non-enveloped, icosahedral, double stranded DNA viruses with a diameter of 45 to 55 nm. They infect epithelial tissues throughout the body leading to both benign and malignant lesions, including common and genital warts. Papillomas caused by some types, such as human papillomaviruses 16 and 18, are strongly associated with cervical cancer. The control contains DNA from HeLa cells that are reported to bear human papillomavirus type 18 sequences.
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Autoimmune Hepatitis(HepAK 7 plus Dot)
Autoimmune Hepatitis(HepAK 7 plus Dot)
  • HepAK 7plus Dot is used for the qualitative determination of IgG autoantibodies to M2, gp210, sp100, LKM1, LC1, SLA and F-actin in human serum or plasma for the differential diagnosis of autoimmune liver diseases.
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PAPP-A
PAPP-A
  • Immunoenzymatic colorimetric method for quantitative determination of PAPP-A (Pregnancy Associated Plasma Protein A) concentration in human serum. PAPP-A kit is intended for laboratory use only.
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Influenza A H1N1
Influenza A H1N1
  • Influenza viruses are enveloped, helical, single stranded RNA (-) viruses with diameters of 80 to 120 nm. Infection typically causes a febrile respiratory illness accompanied by systemic symptoms.
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TOXOPLASMA GONDII DNA Ctrl
TOXOPLASMA GONDII DNA Ctrl
  • Toxoplasma gondii is a protozoan that presents three stages in its developmental life cycle: oocyst, tachyzoite and bradyzoite. While only members of the family Felidae can be definitive hosts of the parasite, a great variety of animals can harbour the tissue cysts. Although consuming contaminated food is the most common way of becoming infected, transplacental infection may occur. The disease is normally benign, but central nervous system disease may appear in immunocompromised patients and the newborn.
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ENTEROCOCCUS FAECALIS (vanB) DNA Ctrl
ENTEROCOCCUS FAECALIS (vanB) DNA Ctrl
  • Bacteria in the genus Enteroccoccus are non-spore forming, facultatively anaerobic, Gram-positive cocci that often occur in pairs or short chains. E. faecalis are non-motile enterococci that can be commonly found as commensal organisms in the intestines of humans. They are able to survive in low-enriched environments such as water, soil or food. Enterococci have both an intrinsic and acquired resistance to antibiotics, making them important nosocomial pathogens, with the ability to cause life-threatening infections in humans. Infections commonly caused by enterococci include urinary tract infections, endocarditis, bacteremia, catheter-related infections, wound infections, and intra-abdominal and pelvic infections. Many infecting strains originate from the patient's intestinal flora.
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Cytobead CeliAK
Cytobead CeliAK
  • CytoBead® CeliAK is a reagent set for the qualitative and semiquantitative determination of endomysial antibodies (EmA) of the IgA and IgG classes, using sections of monkey esophagus, as well as the determination of IgA/IgG antibodies against tissue transglutaminase (tTG; Transglutaminase 2), deamidated gliadin (DG) and a control of IgA antibodies in human serum.
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Rubella IgG
Rubella IgG
  • Rubella is an exanthematous viral disease of children and young adults. It is a self-limited and benign disease characterized by fever, mild upper respiratory symptoms, erythematous rash and suboccipital lymphodenopathy. Rubella can be a very serious disease early in pregnancy leading miscarriages or birth defects up to 85% of cases. Reinfection occurs more frecuently in vaccinated that in naturally immune individuals. The majority of these reinfections occur without symptoms. Rubella reinfections during pregnancy rarely results in transmission of the virus to the unborn child. Antibodies appear at the begining of the disease and initially both IgG and IgM can be detected. IgG antibodies usually persist throughout life. IgM antibodies do not persist beyond 8 weeks. The RUBELLA ELISA IgG has been standardised against the WHO first international standard for anti-rubella immunoglobulin with a cut off control set at 10 U.I./ml
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AMPLIRUN® YELLOW FEVER VIRUS RNA CONTROL
AMPLIRUN® YELLOW FEVER VIRUS RNA CONTROL
  • Yellow fever virus belongs to the family Flaviviridae which includes enveloped, icosahedral, single stranded RNA (+) viruses with a diameter of approximately 50 nm. The infection can occur in one or two phases. The acute phase usually causes fever, muscle pain with prominent backache, headache, shivers, loss of appetite, and nausea or vomiting. Most patients recover after 3 to 4 days, but 15% of them enter a second, more toxic phase with high fever, jaundice, abdominal pain with vomiting and bleeding. The mosquito is the primary vector and carries the virus from one host to another, primarily between monkeys, from monkeys to humans, and from person to person.
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CRYPTOCOCCUS NEOFORMANS DNA Ctrl
CRYPTOCOCCUS NEOFORMANS DNA Ctrl
  • Cryptococcus neoformans is a round to somewhat oval yeastlike fungus with single budding, belonging to the Phylum Basidiomycota, that ranges greatly in size (3.5-8 μm). The yeast is commonly associated to pigeon droppings, and, after inhalation, may spread to the brain and meninges, specially in patients with altered humoral and cellular immunity.
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CANDIDA ALBICANS IFA IgG
CANDIDA ALBICANS IFA IgG
  • Invasive candidiasis is a disease of fungal ethiology with an increasing incidence, specially in immunosuppressed patients (graft receivers, neutropenic and AIDS patients, etc), long-stay hospitalized and catheterized patients, as well as those subjected to extense surgery or receiving broad spectrum antibiotic therapy. The diagnosis of invasive candidiasis is specially difficult due to the absence of pathognomonic symptoms specific of the disease and the low recovery of the microorganism in culture. The present commercial diagnostic techniques show a low specifity and sensitivity, being some of them too difficult to be carried out in a Clinical microbiology laboratory. In order to overcome these problems, a technique for the serologic diagnosis of invasive candidiasis has been developed. This test is based upon the detection of specific antibodies against antigens located on the cell wall surface of the micelium of Candida albicans. These antibodies are normally present in sera from patients with invasive candidiasis caused by C. albicans and other species of this genus. The assay is performed by indirect immunofluorescence after removal of other anti-candida antibodies usually found in most human serum, thus avoiding possible false positive results. This test can be performed with equipment available in a clinical laboratoy and is completed in 3 hours.
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Helicobacter Pylori IgA
Helicobacter Pylori IgA
  • Helicobacter pylori has a worldwide distribution and a high prevalence. The infection with H. pylori is well established as a major cause of gastric and duodenal ulcers. The persistent infection with H. pylori is a risk factor for the development of gastric carcinoma and lymphoma. The infection produces elevated levels of specific H. pylori IgG and IgA antibodies in serum. IgM specific levels has not proven useful in the clinical laboratory. ELISA tests for the detection of H. pylori antibodies are sensitive, specific and cost effective in untreated patiens. The detection of H. pylori specific IgA alone is less sensitive than the detection of specific IgG antibodies. In untreated persons specific IgG and IgA remain elevated for years and successful eradication decreases the IgG and IgA levels, although in some individuals specific antibodies can persist during a long time. Because the infection with H. pylori is so prevalent the test should be performed only on individuals with symptoms. The prevalence of H. pylori antibodies increases with the age. A positive result only indicates that the patient has antibodies to H. pylori and if the individual has not been treated, a positive result very likely indicates an active infection with H. pylori. A definitive diagnosis should be given only when the clinical signs and symptoms of the patient are compatible.